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Original

To explore the behavioral and functional consequences of disturbing the intracellular Ca2+-homeostasis, we have studied genetically knocked-down flies for the GABABR2 (RNAi) specifically in the ORNs, as well as blocking one component of it signaling pathway, the G-protein, by the pertussis toxin. These both groups of fly present strong behavioral deficit adaptation-disrupted flies are not able to discern between odors versus air after a 5-min of exposure to odor (Figure 8).

After Proofreading

To explore the behavioral and functional consequences of disturbing intracellular Ca2+-homeostasis, we have studied flies with GABABR2 genetically knocked-down specifically in the ORNs (RNAi), as well as flies with a component of the signaling pathway, the G-protein, blocked by the pertussis toxin. Both these groups of flies present strong behavioral deficits and are adaptation-disrupted, as they are not able to discern between odors and air after a 5-min exposure to odor (Figure 8).

After Critical Editing

To explore the behavioral and functional consequences of disturbing intracellular Ca2+-homeostasis, we have studied flies with GABABR2 specifically depleted in ORNs by RNAi. We have also examined flies with a key component of the signaling pathway, the G-protein, blocked by the pertussis toxin. Both these groups of flies have strong behavioral deficits. In particular, they are unable to distinguish odors and plain air after a 5-min odor exposure, and are therefore adaptation-disrupted (Figure 8).

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